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In this case, the limiting factor is the maximum bit rate in the downlink direction, which limits the reachable throughput to 16 kb/s. Tests results are depicted in Figure 10.19. The TCP throughput measured at the client shows clearly the e ects of the tra c management functionalities of the GGSN. These mechanisms are implemented for each PDP context, so the correct association between APN and QoS pro le parameters is fundamental, since it could make ine ective all prioritisation mechanisms in the end-to-end chain. As described in 6, the GGSN is the edge element for GPRS and UMTS packet data services. Depending on the vendor s implementation choices, the vendor can be aware of the di erent services requested by the users, since it can perform inspection and analysis of the applications carried by IP packets. In this scenario the key element is the access point name (APN), which de nes the entry to external packet data networks (both Internet and private networks). (The APN is a label which according to DNS naming

does not yield a line image in any plane. However, it is referred to as the x or the 0 astigmatism in conformance with the primary x astigmatism, because of its variation with y as cos 2y. Similarly, the other forms of astigmatism are referred to as indicated in Table 13.2. The Zernike coma terms a8 Z8 r; y and a7 Z7 r; y are called the x and y Zernike comas. They yield PSFs that are symmetric about the x and y axes, respectively. Similarly, the names for the secondary and tertiary coma can be explained. It should be evident that when the cosine and sine terms of a certain aberration are present simultaneously, as in optical testing, their combination represents the aberration whose orientation depends on the value of the component terms (Wyant and Creath, 1992; Mahajan and Swantner, 2006). For example, if both x and y Zernike tilts are present in the form W r; y a2 Z2 r; y a3 Z3 r; y 2a2 r cos y 2a3 r sin y 13:60a

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Summary . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 328

resistance, glucose intolerance and ultimately type 2 diabetes (Wang et al., 2005; Boyko et al., 2000; 10). In turn, both obesity and type 2 diabetes increase cardiovascular risk (Haffner et al., 1998; Tominaga et al., 1999). The presence of type 2 diabetes therefore confers high risk on the obese patient, making it important to ascertain the glycaemic status in all cases. Diabetes can be diagnosed from a raised fasting plasma glucose concentration ( 7.0 mmol/l) and/or from an elevated plasma glucose ( 11.1 mmol/l) 2 hours after a 75-g oral glucose tolerance test (OGTT) (Table 15.4). A fasting plasma glucose in the range 6.1 6.9 mmol/l identifies the condition of impaired fasting glucose (IFG), which although in the sub-diabetic range, confers increased risk. In addition, a 2-hour post-OGTT plasma glucose of 7.8 11.0 mmol/l indicates impaired glucose tolerance, which carries increased cardiovascular risk as well as a 30 50% chance of progressing to type 2 diabetes within a few years (Tominaga et al., 1999; Unwin et al., 2002; Nathan et al., 2006). Thus, both tests may be needed to define an individual s glycaemic status. Fasting glucose, the preferred test to detect diabetes, should be measured first, with non-diabetic subjects then proceeding on another day to an OGTT (American Diabetes Association, 2007).

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Perusse, L, Tremblay, A, Leblanc, C et al (1988) Familial resemblance in energy intake: contribution of genetic and environmental factors The American Journal of Clinical Nutrition, 47 (4), 629 35 Perusse, L, Rankinen, T, Zuberi, A, Chagnon, YC, Weisnagel, SJ, Argyropoulos, G, Walts, B, Snyder, EE and Bouchard, C (2005) The Human obesity gene map: the 2004 update Obesity research, 13, 381 490 Peters, T, Ausmeier, K, Dildrop, R and Ruther, U (2002) The mouse Fused toes (Ft) mutation is the result of a 16-Mb deletion including the entire Iroquois B gene cluster Mammalian genome 13, 186 8 Prentice, AM, Black, AE, Coward, WA et al (1986) High levels of energy expenditure in obese women British Medical Journal (Clinical research edition), 292, 983 7 Prentice, AM and Jebb, S.

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